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Abstract
Polycystic ovary syndrome (PCOS) is characterized by increased luteinizing hormone (LH) secretion, driving excess androgen production. Recent studies show that anti-mullerian hormone (AMH) production is also elevated in PCOS and serum AMH levels positively correlate with LH and androgen levels. In vitro data suggest a causal relationship: Stimulation with LH significantly increases AMH secretion from granulosa cells of PCOS but not of normal women. We investigate the possibility of a direct effect of LH on AMH production in vivo by assessing AMH and androgen responses after human chorionic gonadotropin (hCG) stimulation (as a surrogate for LH) in PCOS and normal women.
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