Abstract
Mitochondrial Permeability Transition (PT) refers to the phenomenon of stress-induced increase in permeability of the inner mitochondrial membrane caused by the opening of the nonspecific channel PT pore. Adenine Nucleotide Translator (ANT) is an important component of PT. However, the precise role of ANT remains controversial with some researchers proposing its primary role in pore formation while others suggesting modulatory role. We hypothesized that ANT involvement in PT might depend on the specific pathway of PT activation.
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