Abstract
We present a case of Retinal Cavernous Haemangioma treated with Intravitreal Bevacizumab, which was initially labelled as persistent proliferative diabetic retinopathy with multiple episodes of vitreous haemorrhage. These lesions can be confused with new retinal vessels in diabetics and if correctly diagnosed unnecessary photocoagulation can be avoided. Our patient received a course of three intravitreal Bevacizumab injections (1.25 mg/0.05 ml) in order to stop the leakage from the retinal cavernous haemangioma lesions and prevent another episode of vitreous haemorrhage. No intraoperative or postoperative complications were seen. Twenty-two months following treatment there was no recurrence of vitreous haemorrhage.
Highlights
IntroductionWe would like to report a case of Retinal Cavernous Haemangioma treated with Intravitreal Bevacizumab, which was initially diagnosed as persistent proliferative diabetic retinopathy with multiple episodes of vitreous haemorrhage
We present a case of Retinal Cavernous Haemangioma treated with Intravitreal Bevacizumab, which was initially labelled as persistent proliferative diabetic retinopathy with multiple episodes of vitreous haemorrhage
Our patient received a course of three intravitreal Bevacizumab injections (1.25 mg/0.05 ml) in order to stop the leakage from the retinal cavernous haemangioma lesions and prevent another episode of vitreous haemorrhage
Summary
We would like to report a case of Retinal Cavernous Haemangioma treated with Intravitreal Bevacizumab, which was initially diagnosed as persistent proliferative diabetic retinopathy with multiple episodes of vitreous haemorrhage. A 59 year-old insulin dependent diabetic male was referred from another ophthalmic unit with a diagnosis of persistent retinal neovascularisation in the left eye, despite repeat sessions of laser photocoagulation. He had a history of multiple mild to moderate self-limiting vitreous haemorrhages in the past which settled without any surgical intervention. Fundus examination showed a cluster of abnormal vessels in the juxtapapillary region of the temporal retina (Figure 1(a)). During that period the patient develop no further episodes of vitreous haemorrhage
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