Abstract
AII is involved in the control of neonatal renal hemodynamics and has been shown to interact with nitric oxide (NO) according to a specific developmental pattern. Distribution of AII receptor AT1 and AT2 subtypes in the kidney is developmentally regulated. The present study examines the effect of AT1 and AT2 receptors specific antagonists on AII/NO interactions in the in vitro, developing rabbit isolated perfused kidney (IPK).
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