Abstract
We read with great interest the article by Dr Nautiyal et al1 dealing with the relationship between angiotensin (Ang)-(1-7) and vagal function in hypertension. The results of their study demonstrated that intracerebroventricular administration of Ang-(1-7) enhanced vagal components of the baroreceptor reflex sensitivity (BRS) for control of heart rate and heart rate variability, independent of altering blood pressure in hypertensive (mRen2)27 rats. Ang-(1-7) did not alter circulating metabolic hormones, nicotinamide adenine dinucleotide phosphate (NADPH) oxidase, or mitogen-activated protein kinase. In …
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