Abstract
THE occurrence of premature ventricular beats during an attack of angina pectoris was first demonstrated by Mackenzie1 with pulse tracings and by Goldhammer and Scherf2 electrocardiographically. Proger, Minnich and Magendantz,3 in a study of the circulatory response of patients with angina pectoris to exercise, showed that one group developed premature beats during attacks and that attacks could be abolished and the exercise tolerance increased by quinidine. This action of quinidine was confirmed by Riseman and Brown,4 who further established the value of this drug in a large group of patients with angina pectoris. In 1948 Porter5 reported 4 cases of . . .
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