Abstract

Echinococcosis granulosus is the leading cause of spinal hydatid disease. Hydatidosis of the bone happens in 0.5%-3% of all the cases: the involvement in the vertebral column is 50%. In the endemic areas, one of the common causes of spinal cord compression is hydatid disease, and the diagnosis may remain obscure until specifying symptoms ensuing from complications due to root and cord compression. We present a case of recurrent spinal cords hydatid cyst in a 44 years old patient because it occurs rarely, and anesthetic management in such cases has never evaluated before.

Highlights

  • Hydatid cysts’ infestation of humans caused by Echinococcosis granulosus or, more rarely, Echinococcus multilocularis.[1]

  • A person who comes in direct contact with the feces of an infected dog or consumes food and milk contaminated by dog feces, when eggs from the tapeworm passed in the feces (The fecal-oral route) may develop hydatid disease.[1,3]

  • One of the expected causes of spinal cord compression is hydatid disease, and the diagnosis may remain obscure until symptoms ensuing from complications due to root and cord compression detect.[4,5]

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Summary

Introduction

Hydatid cysts’ infestation of humans caused by Echinococcosis granulosus or, more rarely, Echinococcus multilocularis.[1]. Echinococcus granulosus is the leading cause of spinal hydatid disease. The spinal involvements usually caused by direct extension from pulmonary or abdominal invasions and rarely begin primarily in the vertebral body. Case Report A male 44 years old (80 kg, 174 cm) was admitted to the neurosurgery ward with complaints of recurrent spinal cords hydatid cyst. Neurological examination revealed spastic paraparesis and hypoaesthesia Anesthetic Implications The patient was scheduled for surgery to excise the cysts and had a laminectomy performed through the posterior approach for neurologic decompression at the level of spinal involvement under general anesthesia. All the cysts extirpated without rupture (Figure 2) Scolicidal solutions, such as hypertonic saline and hydrogen peroxide, were not used during surgery to prevent chemical injury to the cord. The postoperative period was fine, and the patient discharged after 5 days without any neurological deficit

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