Abstract
MOvergrowth of Candida tends to produce high levels of secondary metabolites affecting the immersion of infectious and degenerative diseases. Biofilm’s existence as a virulence factor of Candida makes it challenging to overcome causing multidrugresistant issues. Studies on the effectiveness of Candida antibiofilm drug candidates should be supported by data related to model structure and molecular interaction within the eradication process of biofilm through homology modeling and in-silico docking. This study aims to determine molecular interactions between 1,3-β-glucanase Achatina fulica in which the substrate is, through homology modeling and docking studies within the biofilm matrix eradication process.
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