Abstract
Chronic infection with human hepatitis B virus (HBV) causes about 887.000 deaths annually and is a major risk factor for liver cancer through as-yet unclear mechanisms. HBV DNA integrates into the host cell genome early during infection. HBV integration is not required for viral replication but contributes to subviral particle formation. It is also associated with HBV-induced liver cancer, so its detection, quantification, and characterization are key to understanding HBV-associated disease.
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Schedule a callMore From: Biospektrum : Zeitschrift der Gesellschaft fur Biologishe Chemie (GBCH) und der Vereinigung fur Allgemeine und Angewandte Mikrobiologie (VAAM)
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