Abstract
The oomycetes, commonly known as water moulds and phylogenetically belonging to stramenopiles, can infect a wide range of plants and animals, with some members eliciting the most destructive plant diseases and posing severe threats to global food security and natural ecosystems (Thines, 2018). For example, Phytophthora infestans causes potato late blight and triggered the Irish Famine in 1840s and P. sojae damages soybean production worldwide. Breeding resistant cultivars is the most efficient measure for managing the diseases instigated by oomycetes. But the success of the endeavor requires a sound and deep understanding of the molecular basis underlying pathogen-host interactions. Recently, Sun et al. (2022) elucidated the mechanism underlying the recognition of XEG1, a key virulent effector of P. sojae, by host immune receptor through combining genetic, molecular and structural approaches, which represents a significant step forward for deeply understanding the infection biology and host immune responses in oomycete diseases.
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