Abstract
Eriskin Still Hastaligi’nin etiyolojisi tam olarak bilinmemekle birlikte degisik enfeksiyoz ajanlarin ve genetik faktorlerin varligi ileri surulmektedir.[3] Still hastalarinda HLA DR2, DR4, DR7 ve HLA B35 pozitifligi sikca gorulmektedir.[4] T hucrelerinin patogenezde rolu oldugunu ileri suren bazi calismalar da vardir.[5] Muhammet Emin ERDEM, Seydahmet AKIN, Asli GOCEK OZAL, Adnan HELVACI, Selahattin ERTURK, Mustafa TEKCE, Mehmet ALIUSTAOĞLU
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Schedule a callMore From: The Journal of Kartal Training and Research Hospital
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