Abstract
As a well-known common neurodegenerative disease in aging, Alzheimers disease will cause the loss of the ability to talk, cognization and memorize. Amyloid beta is considered to be a major cause of Alzheimers disease. It will form many different types and accumulate in human brain and cause Alzheimers disease. However, a recent report about the plagiarism of the fundamental paper of this theory makes many people think that our research direction on Alzheimers disease is wrong. The author agrees that the plagiarism of the paper is terrible and should be punished, but the author does not think that this will waste everything we have done about amyloid beta. Amyloid beta is still a major reason that causes Alzheimers disease and find a way to stop the accumulation of amyloid beta in brain is a feasible way to cure Alzheimers disease. Research on the role of amyloid beta in Alzheimers disease, whether it is a major cause is the major purpose of the essay. Whats more a new method that aim to reduce amyloid beta in brain to cure and relief Alzheimers disease will be introduced in this article. This method aims to stop the signal transduction of amyloid beta to neuron cells, thus, stop amyloid beta from damaging our brain and cause Alzheimers disease. The reason this article is written is to confirm the importance of amyloid beta for Alzheimers disease. In addition, introduce a new cure method. Since this method is new, conclude the advantages and disadvantages of this method could inspire other researchers, and improve the performance of the drugs developed based on this method to reach the final goal, which is cure the Alzheimers disease.
Published Version (Free)
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Disclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.