Abstract
During hypo-osmotic stress, the amino-acid concentration of isolated axons of C. sapidus decreases while the specific antivity of these compounds labelled from glucose increases. On the contrary, the amino-acid content increases and there is no significant change in specific activity during hyperosmotic shock. These results are discussed in terms of the mechanisms involved in the control of the amino-acid pool in the process of isosmotic intracellular regulation.
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