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Abstract
Alteration in Antioxidant Defense System and Oxidative Stress in blood for Non Alcoholic Fatty Liver Disease
Highlights
Non-alcoholic fatty liver disease (NAFLD) is defined as steatosis affecting >5% of hepatocytes in the absence of excessive significant alcohol consumption
Anthropometric assessment, clinical parameters and biochemical parameters In this cross-sectional study, controls and NAFLD participants were studied for anthropometric assessment, clinical parameters and biochemical parameters
Our study indicates presence of metabolic GR activity for NAFLD participants compared to controls syndrome such as high BMI, dyslipidaemia or/and elevated [18]
Summary
Non-alcoholic fatty liver disease (NAFLD) is defined as steatosis affecting >5% of hepatocytes in the absence of excessive significant alcohol consumption. The histological spectrum of NAFLD includes non-alcoholic fatty liver (NAFL; steatosis without hepatocellular injury), steatohepatitis (NASH; steatosis with inflammation and hepatocyte ballooning degeneration), fibrosis and . SGOT: serum glutamic oxaloacetic transaminase GGT: Υ- glutamyl transferase ALP: alkaline phosphatase GAE: gallic acid equivalent TE: trolox equivalent ROS: reactive oxygen species HCC: hepatocellular carcinoma cirrhosis [1]. NAFLD has become the most common cause of chronic liver disease worldwide in the last three decades and a major cause of liver- related morbidity and mortality. Clinical burden of NAFLD is confined to liverrelated morbidity and mortality, but there is a growing evidence that NAFLD is a multi-system disease, affecting several extra-hepatic organs like kidney, lung and heart etc.
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