Abstract
In neonatal (A x B) F1 mice, injections of maternal (A strain) antibody to-the Ig allotype of the paternal (B) strain chronically suppress theproduction of antibodies with the B-strain allotype. Here Leonore Herzenberg describes how, in such animals, this form of suppression influences the control of antibody responses to a thymus-dependent antigen that is subsequently encountered.
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