Abstract
Aims: Activin A, a multifunctional regulatory protein of the TGF- β family, gathers more and more attention as a potential neuroprotective agent [2]. Increased cerebral expression of activin A has been observed in animal models after hypoxic-ischemic insults. Of note, neuroprotective effects of exogenous activin after hypoxic-ischemic and kainat-induced injury have been shown in the adult rodent brain [1]. In addition, elevated serum activin A levels have been observed in asphyxiated newborns. The present study investigated mechanisms of hypoxic regulation of activin A in developing mouse brain.
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