Abstract
The local O2 supply of liver tissue during the development of hemorrhagic shock was investigated with O2 electrodes. It was found that local anoxia and disturbances in the microcirculation could be detected even in the early phase of hemorrhagic shock, when systolic blood pressure was still at or above 100 mm Hg. Fig 1 shows a typical example of early tissue anoxia in dog liver.KeywordsHemorrhagic ShockAnaerobic GlycolysisPyridine NucleotideLactate FormationGlucose LactateThese keywords were added by machine and not by the authors. This process is experimental and the keywords may be updated as the learning algorithm improves.
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