Abstract
It has been established that acylhydroperoxy derivatives of phospholipids of oxidized rat liver mitochondria during co-incubation with blood plasma lipoproteins are captured predominantly by LDL particles but not by HDL, which refutes the previously stated hypothesis about the involvement of HDL in the reverse transport of oxidized phospholipids and confirms the possibility of different mechanisms of lipohydroperoxide accumulation in LDL during oxidative stress.
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