Abstract
Intermittent theta-bust stimulation (iTBS) is emerging as a first-choice transcranial magnetic stimulation (TMS) protocol in the treatment of refractory Major Depressive Disorder (MDD). While the mechanisms for antidepressant efficacy remain unclear, physiological studies suggest that N-Methyl-D-Aspartate (NMDA) receptor activation is required for iTBS related synaptic plasticity. No study to date has directly targeted the NMDA receptor as a means of enhancing clinical efficacy in MDD.
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