Abstract

The claim that addiction is a brain disease is almost universally accepted among scientists who work on addiction. The claim’s attraction rests on two grounds: the fact that addiction seems to be characterized by dysfunction in specific neural pathways and the fact that the claim seems to the compassionate response to people who are suffering. I argue that neural dysfunction is not sufficient for disease: something is a brain disease only when neural dysfunction is sufficient for impairment. I claim that the neural dysfunction that is characteristic of addiction is not sufficient for impairment, because people who suffer from that dysfunction are impaired, sufficiently to count as diseased, only given certain features of their context. Hence addiction is not a brain disease (though it is often a disease, and it may always involve brain dysfunction). I argue that accepting that addiction is not a brain disease does not entail a moralizing attitude toward people who suffer as a result of addiction; if anything, it allows for a more compassionate, and more effective, response to addiction.

Highlights

  • Reviewed by: Richard Holton, Massachusetts Institute of Technology, USA Jerome C

  • The claim that addiction is a brain disease is almost universally accepted among scientists who work on addiction

  • The claim’s attraction rests on two grounds: the fact that addiction seems to be characterized by dysfunction in specific neural pathways and the fact that the claim seems to the compassionate response to people who are suffering

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Summary

Introduction

I will argue that though there is a case for saying that the correlates of addiction are pathological, these correlates are not sufficient for the person to have a disease in some accessible environments. Addiction is a brain disease only if pathological deviations from norms of brain function are (in almost any accessible environment) sufficient for being impaired.

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