Abstract
Renal dysfunction due to hypercalcemia typically manifests with impairment in the ability to concentrate urine, decreased glomerular filtration rate (GFR), natriuresis, nephrocalcinosis and diabetes insipidus. Acute Tubular Necrosis (ATN) is a rare manifestation of severe hypercalcemia (defined as serum calcium greater than 20 mg/dl). We present a case of severe hypercalcemia resulting from Milk-Alkali Syndrome caused by chronic ingestion of calcium-based antacids (TUMS) resulting in Acute Kidney Injury (AKI) from ATN.
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