Abstract
In recent years, considerable improvement in immediate postoperative cadaver kidney allograft function has been observed. This is the result of several factors, particularly more adequate recipient selection, better preoperative preparation, improvement in graft preservation, improved perioperative management in operative techniques and more efficient immunosuppression [1]. Nevertheless, graft dysfunction in the first weeks after transplantation is observed in many patients and only approximately 30% of the patients have no episode whatsoever of graft dysfunction by the first year post-transplantation. As the number of acute rejection episodes has decreased, particularly after introduction of cyclosporin A, the relative contribution of other causes of renal dysfunction has relatively increased.
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