Abstract
Trigeminal sensory neuropathy often occurs in association with Sjogren syndrome, sarcoidosis and inflammation, but these diseases were unlikely in this case. The patient was thought to have idiopathic trigeminal sensory neuropathy (ITSN), which may be post-infectious. 3DFlash MRI, an advanced T1-weighted image, showed no gadolinium enhancing lesion (Fig. 1A, arrow). By contrast, CISS MRI, an advanced method of T2*-weighted imaging, showed a high signal intensity lesion (Fig. 1B, arrow), which disappeared after treatment with corticosteroids. Although a few cases of ITSN have been reported to demonstrate gadolinium enhancement lesions, it is generally difficult to detect intraneural lesions using conventional MRI. Because the high signal intensity lesion in CISS MRI could not be detected by 3D-Flash MRI, even with Gd enhancement, the lesion may have repre-
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