Abstract
The continued inability to influence the outcome of acute hemorrhagic pancreatitis in any significant fashion by pharmacologic agents led us to study another approach to altering this often lethal process. In 1967, Andreadis and associates (1} induced pancreatitis in dogs by infusing a mixture of blood, trypsin, and bile into the pancreatic duct under 200 mmHg pressure. All control animals died in shock within 4 to 16 hours. Treatment with vasopressin increased their survival time. Andreadis and associates (1), however, had caused hemorrhagic pancreatitis by rupturing the pancreatic ducts. Our study was undertaken to study the effects of vasopressin on canine hemorrhagic pancreatitis experimentally induced by the closed loop technic (2).
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