Abstract
Conjugate eye movements are controlled via a complex circuit including the inferior parietal lobule, frontal eye field (FEF), dorsolateral prefrontal cortex, superior colliculus, cerebellum, as well as oculomotor and vestibular nuclei in the brainstem.1 Various lesions within this network may cause abnormalities of gaze; we present a case demonstrating the challenges of localizing the causal site for acute conjugate eye deviation (CED).
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