Acute exotropia and blepharospasm due to bilateral paramedian thalamic infarct

Abstract

of the vergence in structures contributing to neural integration may be due to disordered convergence secondary to organic conditions. Although the anatomic pathway of these signals has not yet been identified, divergence eye movements have been assumed to result from a complementary input from divergence neurons to the lateral rectus motoneurons. Divergence position in our case might be related to convergence paralysis or excessive stimulation of divergence neurons due to involvement of vergence pathways. Data from previous studies indicate that convergence paralysis might be more responsible for acute exotropia in bilateral paramedian thalamic lesions.