Abstract
El~~~on~lsive therapy (ECT) in humans does not appear to result in consistent changes in cerebrospinal fluid (CSF) levels of the major monoamine metabolites (Lerer 1987). Some studies reported no change in CSF homovanillic acid (HVA), and 5hydroxyindoleacetic acid (5 HIAA) following the ECT course (Abrams et al. 1976), whereas others have reported a reduction in the probenecid-induced accumulation of both HVA and 5HIAA (Jori et al. 1975), and a recent study found elevation in CSF HVA levels from pre-ECT to I week
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