Abstract
Rationale: Several data support a role of Activin A (ActA) in cancer-induced cachexia (CC). Circulating ActA levels are elevated in patients with CC and predictive of mortality (1-2). However, the mechanisms through which ActA could mediate CC and in particular skeletal muscle atrophy in humans are not yet fully understood. In this work, we aimed to investigate the effects of ActA on human skeletal muscle.
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