Abstract
Abnormal endothelial cell (EC)/trophoblast (TB) interactions are associated with improper implantation, early pregnancy failure and poor placentation. Notch signaling is a key regulator of angiogenesis. Our goal was to define the cell types with Notch activity and correlate this with Notch protein and ligand expression during uterine decidualization and placentation.
Full Text 
Sign-in/Register to access full text options
Sign-in/Register to access full text options 
Paper version not known
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
