Abstract
RATIONALE: Under healthy conditions, the digestive tract is the only non-hematopoietic organ showing resident eosinophils. Under inflammatory conditions, eosinophilic infiltration occurs in several organs, often leading to tissue damage. Neither the function of resting intestinal eosinophils nor the immunopathogenic mechanisms that drive resting cells to activation is clearly understood. In eosinophilic esophagitis (EE), we hypothesized that the eosinophils infiltrating the esophagus are activated and that the eosinophil population present in the intestine is resting.
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