Abstract

THIOUREA and derivatives1–3 and sulphonamide compounds4,5 inhibit the formation of the catabolic hormone both in normal and in hyperactive thyroids. In a vain attempt to compensate for the lack of hormone, the gland undergoes hyperplasia, and hypothyroidism with hyperplastic goitre can result1,6. A few isolated results suggest that the possibility of the drugs also antagonizing the circulating hormone cannot be excluded7.

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