Abstract
THIOUREA and derivatives1–3 and sulphonamide compounds4,5 inhibit the formation of the catabolic hormone both in normal and in hyperactive thyroids. In a vain attempt to compensate for the lack of hormone, the gland undergoes hyperplasia, and hypothyroidism with hyperplastic goitre can result1,6. A few isolated results suggest that the possibility of the drugs also antagonizing the circulating hormone cannot be excluded7.
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have