Abstract
Action of methyl jasmonate and salt stress on antioxidant system of Arabidopsis plants defective in jasmonate signaling genes
Highlights
A signaling pathway of JA is opened for specific transcription factors MYC2, MYC3 and MYC4 [4]
We showed less effective functio ning of stress-protective systems under conditions of salt stress in Arabidopsis mutant jin1 treated with JA compared to wild-type plants [9]
A number of studies have indicated the involvement of the transcription factor JIN1/MYC2, previously activated by jasmonate signal, in the regulation of abscisic acid (ABA)-dependent salt stress reactions [16]
Summary
A signaling pathway of JA is opened for specific transcription factors MYC2, MYC3 and MYC4 [4]. A number of studies have indicated the involvement of the transcription factor JIN1/MYC2, previously activated by jasmonate signal, in the regulation of ABA-dependent salt stress reactions [16]. Functioning of the antioxidant and osmoprotective systems in the Arabidopsis mutant defective in COI1 jasmonate receptor under salt stress has not yet been studied, there are data indicating its role in regulating biosynthesis of flavonoid compounds, expression of non-specific peroxidase and ascorbate peroxidase genes [18, 19], which are important for adaptation to salinity and other stress factors. In connection with the foregoing, the aim of the study was to compare the functioning of the antioxidant and osmoprotective systems of coi (coronatine insensitive 1) and jin ( jasmonate insensitive 1) mutants, defective in jasmonate signaling, and wildtype Arabidopsis thaliana plants under salt stress in the presence and absence of exogenous methyl jasmonate (MJ)
Sign-in/Register to view highlights & summary Sign-in/Register to access full text options 
Free) 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a call
