Abstract
Acquired ocular motor apraxia is characterized by the inability to initiate intentional saccades. Saccades evoked by vestibular stimulation are present. Bilateral lesions involving the frontal and parietal cortex are hypothesized. The number of clinico-pathological studies is small; lesions are usually extensive.Case report: A 73-year-old woman was reanimated after a cardiac arrest. She rapidly recovered. Consciousness was normal. There was no limb paralysis. Facial and eyelid movements were normal, spontaneous and on command. Pupillary light reflex was present. The eyes were in the midline position and immobile. Intentional saccades, to command and visually guided, were absent in the horizontal and vertical planes. Sporadically, saccade on command elicited a head movement associated with a slow vestibular eye movement in the opposite direction. Smooth pursuit was absent in all directions. Optokinetic stimulation provoked no eye movements. Oculocephalic movements were present. Cold water irrigation of one...
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