Abstract
The blood acid-base balance is strikingly affected by hemodialysis. A restoration of blood buffer systems, (expressed in [HCO 3 −]) is the most prominent change, and since reduction in pCO 2 persists the initial metabolic acidosis is converted to respiratory alkalosis at the end of dialysis. The restoration of [HCO 3 −] occurs without concomitant change in sodium concentration and therefore hemodialysis is applicable to therapy of [H +] excess due to any cause, when such occurs in patients with a limited sodium tolerance. A finding previously not observed was a significant rise in blood lactate and pyruvate concentrations occurring during dialysis.
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