Abstract
Incomplete exclusion of the false lumen after stent-graft (SG) repair of aortic dissection (AD) confers risk of subsequent false lumen enlargement and aortic rupture. Endoleak physiology in AD is complex and poorly understood. We hypothesize that severe aortic curvature and coverage of the left subclavian artery (LSCA) are associated with type 1 and 2 endoleaks, respectively.
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