Abstract
Calcium homeostasis is closely regulated mainly by the parathyroid hormone (PTH) and to a lesser degree by the activated form of vitamin D. Primary hyperparathyroidism and underlying malignancy are the two most common causes of hypercalcemia. Few cases of rebound hypercalcemia have been reported in the recovery period of rhabdomyolysis-induced acute kidney injury (AKI) despite the relatively high incidence from up to one third of cases. Here we report a case of severe and symptomatic hypercalcemia in the recovery period of rhabdomyolysis and AKI with low PTH, PTH-related peptide (PTHrP) and 1,25-dihydroxycholecalciferol levels, and normal 25-hydroxycholecalciferol.
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