Abstract

Various mutagenically-effective treatments for Sh2locus failed to produce any back mutation of the closely linked [Formula: see text] marker complex. Nor were any Ac-like elements generated. An excision-repair model was proposed to explain the stability of Ds mutations in the absence of Ac and its high mutability in the presence of Ac. A number of other observations on mutable loci in maize were also explained on the basis of this scheme.

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