Abstract
Among the non-pathologic determinants of auditory brainstem response (ABR), the body temperature is known to act centrally; a decrease in the body temperature causes a greater delay in the later waves and a prolonged central conduction time. We measured the ABR in acute streptozotocin-diabetic mice to determine the interaction of the body temperature and the metabolic disturbance. The degree of prolongation of the central conduction time was smaller in the diabetic mice when the body temperature was lowered, but the mechanism of this apparent resistance to a change in temperature remains unclear. Thus, caution should be paid to the body temperature in discussing ABR latencies, especially in metabolic disturbances such as diabetes mellitus.
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