Abstract
Psoriasis is a chronic inflammatory disease with heightened cardiometabolic risk and adverse cardiovascular (CV) events (Greb et al., 2016). Patients with psoriasis tend to be overweight and have increased visceral adipose tissue (VAT) volume. VAT, a metabolically active fat depot (Wong et al., 2019) accompanied by immune cell dysfunction, is associated with early signs of coronary plaque progression such as noncalcified burden (NCB) and vascular inflammation (Hjuler et al., 2017; Sajja et al., 2020) beyond traditional CV risk factors.
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