Abstract
Modeling is a beneficial tool for understanding cardiovascular system functioning and pathophysiology of the system. The present study is concerned with left ventricle Pressure-Volume diagrams obtained by modeling of aortic aneurysms using the cardiovascular electronic system. In addition, these graphs were compared with a normal condition. The electrical circuit of the cardiovascular system consists of 42 segments including the main arterial vessels. Anatomical and physiological data for circuit parameters have been extracted from medical articles and textbooks. The pressure-volume graphs were obtained for fusiform and saccular aortic aneurysms at different values by using Matlab software. The results show that hypertension is a major symptom of these diseases. By increasing in amount of aneurysm, the blood pressure increases considerably, moreover, the amount of stroke work could be obtained from the graphs. Also, the diagrams show that the amount of cardiac function in every severity of the diseases.
Highlights
The aortic aneurysm is a dilatation of the aortic wall which occurs in two saccular and fusiform types
Fifty percent of patients who experience a rupture of a thoracic aortic aneurysm die before reaching the hospital
It can be understood that maximum increase in left ventricle pressure is about 161 mm Hg
Summary
The aortic aneurysm is a dilatation of the aortic wall which occurs in two saccular and fusiform types. The abdominal aortic aneurysm is the most common form of the aneurismal disease[1,2] In the former 30 years, the occurrence of Abdominal Aortic Aneurysms (AAA) has increased threefold. Aneurysms which involve the ascending aorta, aortic arch and descending thoracic aorta are termed thoracic aortic aneurysms. Aneurysms in these regions are prone to rupture once they reach a certain size. Fifty percent of patients who experience a rupture of a thoracic aortic aneurysm die before reaching the hospital. We have tried to present the cardiac pressure-volume loops for aortic aneurysms with different diameters by utilizing electronic cardiovascular modeling. Comparing the loops with the relevant experimental and clinical observations in order to confirm the results
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