Abstract

BackgroundJuvenile idiopathic arthritis (JIA) is group of heterogenous diseases [1]. Inflammatory cytokines, like Iiterleukin 6 (IL-6), IL-1 and tumor necrosis factor alpha (TNF-a) and their regulatory gene polymorphisms can affect...

Full Text
Paper version not known

Talk to us

Join us for a 30 min session where you can share your feedback and ask us any queries you have

Schedule a call