Abstract

BackgroundSystemic lupus erythematosus (SLE) is a multisystem autoimmune disease with diverse pathogenesis. Excessive apoptosis and impaired clearance of apoptotic cells are the primary drivers of Interferon I (IFN) signaling pathways....

Full Text
Published version (Free)

Talk to us

Join us for a 30 min session where you can share your feedback and ask us any queries you have

Schedule a call