Abstract
Biomechanical forces play a crucial role in cardiac muscle adaptation and disease. Relevant mechanical elements include tensile, compressive and shear stress. Therefore the hypothesis is that the direction and rate of cyclic mechanical strain regulate protein phosphorylation and localization. Flexcell stretch device administers cyclic strain of desired amplitude and frequency to cultured neonatal rat myocytes oriented by microfabricated grooves to a transverse or longitudinal uniaxial force vector.
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