Abstract

Cranial nerve palsies are potential but rare complications of spinal anaesthesia. Most of the literatures support upper cranial nerve palsies like VI, IV and III cranial nerve palsies. Intrathecal hypotension resulting in tractional injury of the cranial nerves is the likely mechanism of injury. As on date, some cases of unilateral vocal fold paralysis and very little bilateral vocal fold paralysis have been described in case reports. We have described a patient who developed hoarseness and dysphagia 7 days after receiving spinal anaesthesia for fixation of inter-trochanteric fracture femur. The patient was diagnosed with bilateral vocal fold paralysis. He was managed conservatively and exhibited complete spontaneous recovery as has been described in the previously reported cases. Any patient presenting with idiopathic vocal fold paralysis should be enquired about the history of spinal or epidural anaesthesia. If the history is affirmative, then it points towards transient intrathecal hypotension as a potential etiology of the cranial nerve palsy.

Highlights

  • Spinal anaesthesia is a commonly used method of anaesthesia in lower limb and lower abdominal surgeries

  • The incidence of cranial nerve palsy following spinal anaesthesia described in older literature is 1 in 200 to 1 in 1,200 cases [1]

  • Cranial nerve palsies, following spinal anaesthesia are a documented though rare complication

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Summary

Introduction

Spinal anaesthesia is a commonly used method of anaesthesia in lower limb and lower abdominal surgeries. The incidence of cranial nerve palsy following spinal anaesthesia described in older literature is 1 in 200 to 1 in 1,200 cases [1]. Case Summary Our patient was an 84 year of gentleman with long standing type 2 diabetes mellitus, ischemic heart disease with moderate LV systolic dysfunction (EF 48 %) having a right sided inter-trochanteric fracture femur He was posted for dynamic hip screw (DHS) fixation. (2020), «EUREKA: Health Sciences» Number 3 evaluation shortly after the start of the symptoms revealed bilateral vocal fold paralysis in flexible endoscopy. It was followed by CT Scan of Brain and spinal cord which were inconclusive.

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