Abstract
Our group has recently identified 121 novel long non-coding RNAs (lncRNAs) whose dysregulated expression stratifies prostate cancer progression. One of these, PCAT1, was upregulated in aggressive disease, and PCAT1 expression inversely correlated with expression levels of BRCA2, a homologous recombination (HR) repair protein. The purpose of this study was to determine the role of PCAT1 in DNA repair and response to genotoxic stress.
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