Abstract
Aim: Low blood levels of HDL-cholesterol increase the risk of coronary heart disease (CHD). HDL particles are complex macromolecules containing hundreds of lipid species and dozens of proteins and exerting many potentially anti-atherogenic activities. Pathological conditions cause quantitative and qualitative molecular changes in HDL components and, thereby, dysfunction. Main goal of the project is to identify structure-function-disease relationships of HDL with a system biology approach.
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