Abstract
Fifteen correlative biochemical and electroencephalographic studies were done on 13 patients with chronic pulmonary disease. Abnormal resting EEG tracings were observed in 7 patients. Results of the biochemical studies indicated a significant elevation of serum potassium in both groups when compared with normal subjects. No difference was observed in the blood gas, electrolyte and glucose values of the electroencephalographically normal and abnormal patients. Correction of the hypoxia and exaggeration of the hypercapnia did not alter the resting EEG in either group of patients. Clinical evidence suggests that the presence of cerebral vascular disease as a function of age may be a factor in the appearance of the abnormal EEG pattern in patients with chronic hypercapnia and anoxia. The evidence presented suggests an adaptive mechanism in chronic hypercapnia preventing the EEG changes observed in acute hypercapnia.
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