Abstract
A mechanism for the radioluminescence excitation involving radiation-induced mobile defects of the lithium sublattice has been revealed for double lithium orthoborates Li6YxGd1−x B3O9 (x = 0.0, 0.5) doped with a cerium impurity (0.5–1.0 at%). The pulse cathodoluminescence and transient optical absorption decay kinetics for both the single crystals and crystal-fibers have been studied using time-resolved absorption and luminescence spectroscopy techniques upon excitation with a nanosecond electron beam at T = 293 K. A numerical simulation of both processes had been performed. Based on the simulation results we explained the role of radiation defects of the lithium sublattice in increasing the radioluminescence intensity and the creation of inertial components of the luminescence decay kinetics.
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