Abstract
Background: Graft hyperperfusion syndrome (HPS) is a deterrent to transplantation using partial grafts. Numerous risk factors were described but no predictive model exists. Thrombocytopenia early after LDLT mainly occurs secondary to graft/splenic sequestration, a product of Portal vein pressure (PVP). Post-operative Portal vein flow (PVF) monitoring unlike PVP, measures inflow, does not directly reflect sinusoidal pressure and predict graft dysfunction secondary to HPS. This study aims to determine whether post-LDLT platelet count (PC) can serve as a biomarker for PVP and present a predictive model for HPS.
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