Abstract
Genetic factors that predispose individuals to ankylosing spondylitis (AS) are not fully understood. Axial and sacroiliac joint fibrosis are characteristic of AS and the presence of TGFβ1 mRNA in AS sacroiliac joints raised the possibility that this cytokine might be implicated in this fibrosis. We have therefore examined a group of HLA B27 positive AS patients to investigate whether they could be prone to fibrosis based on overproduction of TGFβ1.
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